Varicella (Chickenpox)
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Introduction
- Varicella Zoster virus is Human Herpes Virus 3
- Transmission is by direct contact with the lesions and by the respiratory route
- Initial replication occurs in the nasopharynx and conjunctivae
- After the primary infection, the virus remains dormant in nervous tissue
- Reactivation later in life is typically manifested as Herpes zoster
Clinical features
- Incubation period is 10 - 21 days
- Vesicular eruptions consist of delicate "teardrop" vesicles on an erythematous base
- The eruption starts with faint macules that develop rapidly into vesicles within 24 hours
- Successive fresh crops of vesicles appear for a few days, mainly on the trunk, face, and oral mucosa
- New lesions usually stop appearing by the fifth day; the majority is crusted by the sixth day
- Most disappear in less than 20 days without a scar, except larger and secondarily infected lesions
- Low grade fever
- Malaise
- Headaches
The severity of the disease is age-dependent
- Adults have more severe disease and a greater risk of visceral disease
Differential diagnoses
- Variola minor
- Disseminated zoster in immunosuppressed patients
- Widespread papular urticaria
- Coxsackie and ECHO viruses eruption
Complications
- Secondary bacterial infection
- Pneumonia
- Cerebellar ataxia and encephalitis
- Reye's syndrome
Investigations
- Tzanck smear
- Direct fluorescent antibody (DFA) staining
- Polymerase Chain Reaction (PCR)
Treatment goals
- Relieve itching and treat secondary bacterial infection
- Reduce severity and scarring
Drug treatment
Aciclovir
Adult: 10 mg/kg intravenously three times daily for 7 days in immunocompromised patients
Child: see Herpes zoster
Antihistamine for pruritus
Co-trimoxazole or erythromycin for secondary infection
Notable adverse drug reactions, contraindications and caution
Aciclovir
- Ensure adequate hydration
- Caution in pregnancy and breastfeeding
- May cause nausea, vomiting, dizziness, fatigue pruritus and photosensitivity
Prevention
- Isolate patients from non-immune persons